2005
Distribution and phenotype of Epstein–Barr virus-infected cells in human pharyngeal tonsils
Hudnall SD, Ge Y, Wei L, Yang NP, Wang HQ, Chen T. Distribution and phenotype of Epstein–Barr virus-infected cells in human pharyngeal tonsils. Modern Pathology 2005, 18: 519-527. PMID: 15696119, DOI: 10.1038/modpathol.3800369.Peer-Reviewed Original ResearchMeSH KeywordsAdolescentAdultAntigens, CDB-LymphocytesChildChild, PreschoolEpstein-Barr Virus InfectionsEpstein-Barr Virus Nuclear AntigensFemaleGene Expression Regulation, ViralHerpesvirus 4, HumanHumansImmunohistochemistryImmunophenotypingMaleMiddle AgedPalatine TonsilT-LymphocytesViral Matrix ProteinsViral ProteinsConceptsEpstein-Barr virusEBER-positive cellsT cellsHuman tonsilsNK cellsPlasma cellsEpithelial cellsB cellsBiology of EBVNK/T-cell lymphomaEpstein-Barr virus-infected cellsEBV latent protein EBNA1CD20-positive B cellsCD3-positive T cellsEarly Protein BZLF1Non-B cell typeNatural killer cellsRare T cellsT-cell lymphomaLMP1-positive cellsVirus-infected cellsMost tonsilsPlasmacytoid morphologyKiller cellsInterfollicular zones
2001
Composite nodular lymphocyte-predominance Hodgkin disease and gamma-heavy-chain disease: a case report and review of the literature.
Hudnall S, Alperin J, Petersen J. Composite nodular lymphocyte-predominance Hodgkin disease and gamma-heavy-chain disease: a case report and review of the literature. Archives Of Pathology & Laboratory Medicine 2001, 125: 803-7. PMID: 11371236, DOI: 10.5858/2001-125-0803-cnlphd.Peer-Reviewed Original ResearchConceptsNodular lymphocyte predominance Hodgkin's diseaseLymphocyte predominance Hodgkin's diseaseHodgkin's diseaseChain diseaseAutoimmune hemolytic anemiaB-cell natureB-cell lymphoproliferative processesComplete remissionGraves' diseaseLymph nodesCase reportLymphoproliferative processMonoclonal gammopathyHemolytic anemiaBone marrowUnique caseDiseaseClonal relationshipFurther treatment
2000
Marked Increase in L-Selectin–Negative T Cells in Neonatal Pertussis
Hudnall S, Molina C. Marked Increase in L-Selectin–Negative T Cells in Neonatal Pertussis. American Journal Of Clinical Pathology 2000, 114: 35-40. PMID: 10884797, DOI: 10.1309/banx-8jkm-tuqn-4d6p.Peer-Reviewed Original ResearchConceptsPeripheral blood lymphocytesL-selectin expressionT cellsBlood lymphocytesNormal CD4/CD8 ratioCD4/CD8 ratioNaïve T cellsDetailed immunophenotypeNeonatal pertussisCD8 ratioMarked lymphocytosisNK cellsHealthy newbornsHLA-DRLymphoid tissueCord bloodT lymphocytesPertussis toxinB cellsL-selectinLymphocytesFlow cytometryPertussisMarked increaseDramatic finding
1996
MASSIVE PSEUDOLYMPHOMATOUS PERICARDIAL EFFUSION IN THE POSTTRANSPLANT SETTING MIMICKING POSTTRANSPLANT LYMPHOPROLIFERATIVE DISEASE
Hudnall1 2, Kathleen M, A. G, Yvonne B. MASSIVE PSEUDOLYMPHOMATOUS PERICARDIAL EFFUSION IN THE POSTTRANSPLANT SETTING MIMICKING POSTTRANSPLANT LYMPHOPROLIFERATIVE DISEASE. Transplantation 1996, 61: 1776-1778. PMID: 8685960, DOI: 10.1097/00007890-199606270-00020.Peer-Reviewed Original ResearchConceptsPericardial effusionLymphocytic effusionsLymphoproliferative disordersB-cell lymphoproliferative disordersBenign pericardial effusionCardiac transplant teamPosttransplant lymphoproliferative diseaseCardiac transplant recipientsCell lymphoproliferative disordersImmunosuppressant withdrawalTransplant recipientsTransplant settingLymphoproliferative diseaseTransplant teamOpposite therapyImmunophenotypic dataTransplant processUnusual caseEffusionDetailed histologicDiagnostic distinctionEBVDisorders
1991
Cyclosporin A renders target cells resistant to immune cytolysis
Hudnall S. Cyclosporin A renders target cells resistant to immune cytolysis. European Journal Of Immunology 1991, 21: 221-226. PMID: 1846819, DOI: 10.1002/eji.1830210133.Peer-Reviewed Original ResearchConceptsCsA dose reductionB-cell hyperplasiaB lymphocytesDose reductionCell growth inhibitionNatural killerCell hyperplasiaEarly post-transplant lymphoproliferative disordersHuman organ transplant recipientsPost-transplant lymphoproliferative disorderT cell-dependent responsesAntibody-dependent responsesPost-transplant settingOrgan transplant recipientsTarget cellsEpstein-Barr virusCell-dependent responsesHuman target cellsComplement-mediated cytolysisGrowth inhibitionTransplant recipientsDisease regressionImmune controlNK cellsLymphoproliferative disorders
1987
B‐cell lymphoma in severe combined immunodeficiency not associated with the Epstein–Barr virus
Garcia C, Brown N, Schreck R, Stiehm E, Hudnall S. B‐cell lymphoma in severe combined immunodeficiency not associated with the Epstein–Barr virus. Cancer 1987, 60: 2941-2947. PMID: 2824020, DOI: 10.1002/1097-0142(19871215)60:12<2941::aid-cncr2820601216>3.0.co;2-a.Peer-Reviewed Original ResearchMeSH KeywordsB-LymphocytesFemaleHerpesvirus 4, HumanHumansImmunologic Deficiency SyndromesInfantLymphomaConceptsEpstein-Barr virus nuclear antigenB-cell lymphomaB-cell lymphoproliferative disordersEBV DNASevere combined immunodeficiencyLymphoproliferative disordersMalignant B-cell lymphomaEBV DNA probePrevention of EBVImmunoglobulin heavy chain gene rearrangementB-cell lymphoproliferationEpstein-Barr virusClonal B-cell lymphomaGene rearrangementsHeavy chain gene rearrangementClonal Ig gene rearrangementsChain gene rearrangementAntiviral chemoprophylaxisTransplant patientsSuch prophylaxisImmunodeficient patientsTumor cell DNAAnticomplement immunofluorescenceSitu cytohybridizationPatients