2017
Stomach and Bone
Kitay AM, Geibel JP. Stomach and Bone. Advances In Experimental Medicine And Biology 2017, 1033: 97-131. PMID: 29101653, DOI: 10.1007/978-3-319-66653-2_6.Peer-Reviewed Original ResearchConceptsBone metabolismGastric acid secretionCalcium-sensing receptorVitamin DParathyroid glandsAcid secretionGastrointestinal organsTherapeutic implicationsEndocrine modulatorsCalcium homeostasisDiseaseStomachOrgansMetabolismImportant playersIntermediary organDiverse tissuesDysfunctionCaSRPTHCalcitoninKidneySecretionIntestineReceptors
2010
Insulin Antagonizes Glucose-Induced Gastric Acid Secretion via the AMP Kinase - A Reason for the High Prevalence of Peptic Ulcer Disease and GERD in DM 2?
von Keudell A, Lehmann R, Kopic S, Murek M, Boroumand R, Geibel J. Insulin Antagonizes Glucose-Induced Gastric Acid Secretion via the AMP Kinase - A Reason for the High Prevalence of Peptic Ulcer Disease and GERD in DM 2? The American Journal Of Gastroenterology 2010, 105: s37. DOI: 10.14309/00000434-201010001-00097.Peer-Reviewed Original ResearchToxin Mediated Diarrhea in the 21st Century: The Pathophysiology of Intestinal Ion Transport in the Course of ETEC, V. cholerae and Rotavirus Infection
Kopic S, Geibel JP. Toxin Mediated Diarrhea in the 21st Century: The Pathophysiology of Intestinal Ion Transport in the Course of ETEC, V. cholerae and Rotavirus Infection. Toxins 2010, 2: 2132-2157. PMID: 22069677, PMCID: PMC3153279, DOI: 10.3390/toxins2082132.Peer-Reviewed Original ResearchConceptsEpisodes of diarrheaIntestinal ion transportSpecific therapeutic approachesMajor healthcare concernRotavirus infectionTherapeutic approachesV. choleraeCommon enterotoxinInfectious agentsPathophysiological processesInfectious diseasesDiarrheaHealthcare concernCellular mechanismsLethal effectsPathophysiologyYearsInfectionCholeraeRotavirusDiseaseETEC
2005
Bicarbonate Secretion
Binder HJ, Rajendran V, Sadasivan V, Geibel JP. Bicarbonate Secretion. Journal Of Clinical Gastroenterology 2005, 39: s53-s58. PMID: 15758660, DOI: 10.1097/01.mcg.0000155521.81382.3a.Peer-Reviewed Original Research