2014
Epstein–Barr Virus: Pathogenesis and Host Immune Response
Hudnall S. Epstein–Barr Virus: Pathogenesis and Host Immune Response. 2014, 7-24. DOI: 10.1007/978-1-4939-0870-7_2.Peer-Reviewed Original ResearchEpstein-Barr virusLarge double-stranded DNA genomeDouble-stranded DNA genomeInfected cellsLytic genesAnti-viral immune responsesTerminal repeat unitsCircular episomal formVirion structural componentsLinear DNA moleculesCellular S phaseLytic replicationLatent genesB cellsImmune responseRepeat unitsImmune recognitionReplication programDaughter cellsInternal repeat unitsDNA genomeGrowth signalsIcosahedral capsidTegument proteinsEBV-specific cytotoxic T cells
2000
T‐gamma gene rearrangement and CMV mononucleosis
Mathew P, Hudnall S, Elghetany M, Payne D. T‐gamma gene rearrangement and CMV mononucleosis. American Journal Of Hematology 2000, 66: 64-66. PMID: 11426498, DOI: 10.1002/1096-8652(200101)66:1<64::aid-ajh1013>3.0.co;2-v.Peer-Reviewed Original ResearchMeSH KeywordsAntibodies, ViralBlood CellsBone Marrow CellsClone CellsCytomegalovirusCytomegalovirus InfectionsDiagnosis, DifferentialFemaleGene Rearrangement, gamma-Chain T-Cell Antigen ReceptorHumansImmunoglobulin MImmunophenotypingInfectious MononucleosisLymphoproliferative DisordersMiddle AgedT-Lymphocyte SubsetsViremiaConceptsT gamma gene rearrangementCMV infectionLymphoproliferative diseaseAcute CMV infectionGene rearrangementsAtypical lymphocytosisCMV mononucleosisCMV viremiaNight sweatsIgM serologyClinical featuresLymphoid aggregatesBlood countPeripheral bloodHemolytic anemiaImmune responseBone marrowWeight lossCytomegalovirusInfectionDiseaseMononucleosisViremiaSplenomegalyLymphocytosis